NCLEX RN 1000RN SUMMARY NOTES WELL UPDATED 2021
ABDOMINAL AORTIC ANEURISM
“4-A’s”
Asymptomatic
Abdominal mass
Abdominal pulse
Aches low back
ABDOMINAL DISTENSTION
"5-F's"
Fat
Fluid
Feces
Flatus
Fetus
ACE
...
NCLEX RN 1000RN SUMMARY NOTES WELL UPDATED 2021
ABDOMINAL AORTIC ANEURISM
“4-A’s”
Asymptomatic
Abdominal mass
Abdominal pulse
Aches low back
ABDOMINAL DISTENSTION
"5-F's"
Fat
Fluid
Feces
Flatus
Fetus
ACE INHIBITOR SIDE EFFECTS
"CAPTOPRIL"
Cough
Anaphylaxis
Palpitations
Taste
Orthostatic -↓BP
Potassium - ↑K+
Renal impairment
Impotence
Leukocytosis
ACID-BASE
"ROME" – Respiratory Opposite, Metabolic Equal
ACIDOSIS
» Respiratory (opposite): pH Pco2
» Metabolic (equal): pH HCO3
ALKALOSIS
» Respiratory (opposite): pH Pco2
» Metabolic (equal): pH HCO3
ACIDOSIS/ALKALOSIS
“ROME”
Respiratory Opposite:
pH↑ PCO2↓ = alkalosis
pH↓ PCO2↑ = acidosis
Metabolic Equal:
pH↑ HCO3↑ = Alkalosis
pH↓ HCO3↓ = Acidosis
ACIDOSIS/ALKALOSIS – COMPENSTATION
“RUB MUB”
Respiratory Uses Bicarb
Metabolic Uses Breathing
ALKALOSIS AND ACIDOSIS
AlKalosis has “K” – it’s “K”icking pH up= PH↑
AciDosis has “D” – it’s “D”ropping pH Down = PH↓
ACUTE INFLAMMATION FEATURES
Think: "what a cute pair of SLIPpeRs"to tie acute inflammation to “SLIPR”mnemonic).
“SLIPR”
Swelling
Loss of function
Increased heat
Pain
Redness
ADRENAL GLAND HORMONES
“SSS”
S-sugar (Glucocorticoids)
S-salt (Mineralocorticoids)
S-sex (Androgens)
AIMS for improvement
“PETEES” AIMS
P - Patient centered care
E - Efficient
T - Timely
E - Effective
E - Equitable
S - Safety
ALCOHOL WITHDRAWAL – CLINICAL FEATURES
"HITS"
Hallucinations (visual, tactile)
Increased vital signs & insomnia
Tremens delirium tremens (potentially lethal)
Shakes/Sweat/Seizures/Stomach pain (N/V)
ALCOHOLISM – BEHAVIORAL PROBLEMS
“5-D's”
D- Denial
D- Dependency
D- Demanding
D- Destructive
D- Domineering
ALCOHOLISM OUTCOME
“BAD”
B- Brain Damage
A- Alcoholic Hallucinosis
D- Death
ALDOSTERONE IS REGULATED BY:
"RNA’S"
Renin-angiotensin mechanism
Na concentration in blood
Anp (ANP – atrial natriuretic peptide)
Stress
ALZHEIMER – “5 A’s” to DIAGNOSIS
“5-A's”
Amnesia – loss of memories
Anomia – unable to recall names of everyday objects
Apraxia – unable to perform tasks of movement
Agnosia – inability to process sensory information
Aphasia – disruption with ability to communicate
ANGINA – PRECIPITATING FACTORS
"4-E's"
Eating
Emotion
Exertion (Exercise)
Extreme Temperatures (Hot/Cold weather)
“ANOREXIA” – EATING DISORDER
A-menorrhea delayed
N-o organic factors accounts for weight loss
O-obviously thin but feels FAT
R-refusal to maintain normal body weight
E-epigastric discomfort is common
X-symptoms (peculiar symptoms)
I-intense fears of gaining weight
A-always thinking of foods
ANOREXIA NERVOSA – CLINICAL FEATURES
"ANOREXIC"
A-adolescent women/Amenorrhea
N-GT alimentation (most severe cases)
O-obsession w/ wt. loss/becoming fat though underweight
R-refusal to eat (5% die)
E-electrolyte abnormalities (e.g., K+, cardiac arrhythmia)
X-exercise
I-intelligence often above average/Induced vomiting
C-cathartic use (and diuretic abuse)
ANTICHOLINERGIC CRISIS
Can't see (blurred vision)
Can't spit (dry mouth)
Can't pee (urinary retention)
Can't shit (constipation)
ANTICHOLINERGIC CRISIS – SIGNS
"SLUD"
Salivation
Lacrimation
Urination
Defecation
ANTI-TB DRUGS& SIDE EFFECTS
“RIPES”
Rifampicin – red-orange urine
Isoniazid – peripheral neuritis
Pyrazinamide –increase uric acid
Ethambutol – eye problems
Streptomycin – ototoxic
(use a star as these drugs stain the teeth) – ???
(H-eat, I-nduration) – ???
APGAR SCORING
"APGAR"
Appearance: cyanosis--peripheral, central, none
Pulse: pulse rate
Grimace: response to stimulation
Activity: movement of the baby (muscle tone)
Respiration: respiratory rate
APPENDICITIS - ASSESSMENT
“PAINS”
Pain (RLQ) - pain in RLQ of abdomen
Anorexia - loss of appetite
Increased temperature, WBC (15,000-20,000)
Nausea
Signs (McBurney's, Psoas)
ARTERIAL BLOOD GASES
1. look at pH:
LowpH = acidosis
High pH = alkalosis
2. use “ROME” mnemonic (to determine if its respiratory or metabolic):
Respiratory –Opposite
Metabolic –Equal
ASSESSING CHANGES IN BEHAVIOR
"DEMENTIA"
Drug and alcohol
Eyes and ears
Metabolic and endocrine disorders
Emotional disorders
Neurologic disorders
Tumors and trauma
Infection
Arterial vascular disease
ASTHMA MANAGEMENT
"ASTHMA"
Adrenergics (Albuterol)
Steroids
Theophylline
Hydration (IV)
Mask (Oxygen)
Antibiotics
ASTHMA - MANAGEMENT
Asthma is a spasm of the airways, which causes difficulty breathing.
“ASTHMA”
Adrenergic (Albuterol)
Steroids
Theophylline
Hydration (IV)
Mask (Oxygen)
Antibiotics
ATRIAL FIBRILLATION – NEW ONSET CAUSES
“THE ATRIAL FIBS”
Thyroid
Hypothermia
Embolism (PE)
Alcohol
Trauma (cardiac contusion)
Recent surgery (post CABG)
Ischemia
Atrial enlargement
Lone or idiopathic
Fever, anemia, high-output states
Infarct
Bad valves (mitral stenosis)
Stimulants (cocaine, theo, amphet, caffeine)ATRIOVENTRICULAR VALVES
"LAB RAT"
Left Atrium: Bicuspid
Right Atrium: Tricuspid
ATROPINE
"A goes with B"
Atropine used to treat bradycardia.
BENNER'S MODEL
N - Nickerr's - easy to remember b/c it rhymes with Benner's Novice – strictly able to focus on learning therules, onset of education
A - and - "ad"vanced beginners– distinguish abnormal findings but cannot readily understand significance
C - Comp - Competent – able to handle their pt. load and prioritize situation
P - Planetary – big picture is the key word - Proficiency
E - Experts– Leader/ role model not every nurse becomes one.
BETA 1 AND BETA 2
Beta 1 adrenergic receptors are mostly found in the heart. Beta 2 adrenergic receptors are found in lungs, GI tract, vascular smooth muscle, skeletal muscle, liver. Beta 1 beta blockers act primarily on the heart. Beta 2 beta blockers act primarily on the lungs.
Beta 1: heart
Beta 2: lungs - You have one heart and two lungs
BETA BLOCKERS
B1 Blocks the heart (only have ONE heart)
B2 Blocks the lungs (have TWO lungs)
BETA BLOCKER CONTRAINDICATIONS
"ABCDE"
Asthma
Block (heart block)
COPD
Diabetes mellitus
Electrolyte (hyperkalemia)
BETA BLOCKERS
"You have 1 heart and 2 lungs"
Beta-1 act primarily on heart
Beta-2 act primarily on lungs
BLEEDING PRECAUTIONS
“RANDI”
R- Razor Electric/Blades
A- Aspirin
N- No needles (esp. in small gauge)
D- Do decrease in needle sticks)
I - Injury (Protect from)
BLEEDING PRECAUTIONS
If a patient is taking an anticoagulant to prevent blood clots there is increased risk for bleeding. Be careful with blades when shaving. Do not take aspirin as it interferes with blood clotting and can magnify the effect of the medication. Avoid excess needle sticks and protect the patient from injury.
“RANDI”
Razor Electric/Blades
Aspirin
Needles- small gauge
Decrease needle sticks
Injury (Protect from)
BLOOD FLOW THROUGH HEART VALVES
"Tissue Paper My Ass"
Tricuspid
Pulmonic
Mitral
Aortic
BLOOD GLUCOSE
Symptom Implication(rhyme)
Cold and clammy . . . give hard candy
Hot and dry . . . glucose is high
BLOOD TYPES
Picture type O as a huge circle, like the universe, because they are the universal donor. They can give to everyone.
However, also think of them as the “odd man out” because they can only receive type O as well. They are universally odd, able to give to all but only receive from themselves!
BODY SYSTEMS
"MR DICE RUNS"
Muscle
Respiratory
Digestive
Integumentary
Circulatory
Endocrine
Reproductive
Urinary
Nervous
Skeletal
BRADYCARDIA & LOW BP DRUGS
“IDEA”
I - Isoproterenol
D - Dopamine
E - Epinephrine
A - Atropine Sulfate
BRADYCARDIA & LOW BLOOD PRESSURE DRUGS
This refers to symptomatic bradycardia and hypotension. Bradycardia and hypotension are not necessarily adverse clinical findings requirement treatment in and of themselves.
“IDEA”
Isoproterenol
Dopamine
Epinephrine
Atropine Sulfate
BRADYCARDIA AND HYPOTENSION MEDS
"IDEA"
Isoproterenol
Dopamine
Epinephrine
Atropine Sulfate
BREASTFEEDING ASSESSMENT
"LATCH"
Latch achieved by infant
Audible swallow
Type of nipple
Comfort of mother
Help given to mother with nursing
BRONCHODILATORS
"TO A SIS"
Terbutaline
Orciprenaline
Adrenaline
Salbutamol
Isoprenaline
Salmeterol
“BULIMIA” – EATING DISORDER
B-binge eating
U-under strict dieting
L-lacks control over-eating
I-induced vomiting
M-minimum of two binge eating episodes
I-increased/Persistent concern of body size/shape
A-abuse of diuretics & laxatives
BULIMIA CLIENT FINDINGS
“WASHED”
W-weight loss of 15% of original body weight
A-amenorrhea
S-social withdrawal
H-history of high activity & achievement
E-electrolyte Imbalance
D-depression/ Distorted Body Image
CANCER ASSESSMENT
"CAUTION"
Change in bowel or bladder habits
A sore that doesn't heal
Unusual bleeding or discharge
Thickening or lump
Indigestion or difficulty swallowing
Obvious changes in a wart or mole
Nagging cough or hoarseness
CANCER - EARLY WARNING SIGNS
Cancer can sometimes be difficult to identify, but the earlier it is detected the better chance treatment will be effective.
“CAUTION UP”
Change in bowel or bladder
A lesion that does not heal
Unusual bleeding or discharge
Thickening or lump in breast or elsewhere
Indigestion or difficulty swallowing
Obvious changes in wart or mole
Nagging cough or persistent hoarseness
Unexplained weight loss
Pernicious Anemia
CANCER - INTERVENTIONS
“CANCER”
Comfort
Altered Body Image
Nutrition
Chemotherapy
Evaluate response to meds
Respite for caretakers
CANCERS THAT METASTASIZE IN BONE
"Particular Tumors Love Killing Bone"
Prostate
Thyroid
Lung
Kidney
Breast
CANES AND WALKERS
"COAL" and "WWAL"
Cane
Opposite
Affected
Leg
Walker
With
Affected
Leg
CANES AND WALKERS
“COAL & WWAL”
C- Cane
O- Opposite
A- Affected
L- Leg
W- Walker
W- With
A- Affected
L – Leg
CARDIAC VALVES BLOOD FLOW
This simple mnemonic helps to remember the order in which blood passes through the four heart valves in sequential order.
“Toilet Paper My A**”
Tricuspid
Pulmonic
Mitral
Aortic
CEREBELLAR DAMAGE SYMPTOMS
"VANISHED"
Vertigo
Ataxia
Nystagmus
Intention tremor
Slurred (or staccato) speech
Exaggerated broad based gait
Hypotonic reflexes
Dysdiadochokinesia.
CHF TREATMENT
"UNLOAD FAST"
Upright Position
Nitrates (in low dose)
Lasix
Oxygen
Aminophylline
Digoxin
Fluids (decrease)
Afterload (decrease)
Sodium restriction
Test (Dig level, ABGs, K level)
CHF - TREATMENT
These treatments used in conjunction will help in reducing the symptoms and complications associated with CHF.
“UNLOAD FAST”
U sit Upright
Nitro
Lasix
Oxygen
Aminophylline
Digoxin
Fluids - decrease
Afterload - decrease
Sodium - decrease
Tests: dig level, ABG, K+
CHILD ABUSE/NEGLECT – WARNING SIGNS
“CHILD ABUSE”
C-child’s excessive knowledge on sex & abusive words
H-hair growth in various lengths
I-inconsistent stories from the child & parent/s
L-low self-esteem
D-depression
A-apathy, no emotion
B-bruised
U-unusual injuries
S-serious injuries
E-evidence of old injuries not reported
Wernicke-Korsakoff syndrome (WKS) – (alcohol-associated neurological disorder)
"COAT RACK"
Wernicke's encephalopathy (acute phase) clinical features:
C-confusion
O-ophthalmoplegia
A-ataxia Thiamine is an important aspect of
T-x Korsakoff's psychosis (chronic phase) characteristic findings:
R-retrograde amnesia (recall of some old memories)
A-anterograde amnesia (ability to form new memories)
C-confabulation
K-korsakoff's psychosis
CHLOROQUINE USES (OTHER THAN MALARIA)
"RED LIP"
Rheumatoid arthritis
Extra intestinal amoebiasis
Discoid lupus erythematosus
Lepra reaction
Infectious mononucleosis
Photogenic reactions
CHOLECYSTITIS – RISK FACTORS
“5 F's”
Female
Fat
Forty
Fertile
Fair
CHOLINERGIC CRISIS
"SLUD"
Salivation
Lacrimation
Urination
Defecation
CHOLINERGIC CRISIS - SIGNS AND SYMPTOMS
“SLUDGE”
Salvation
Lacrimation
Urination
Defecation
Gastric upset
Emesis
CHRONIC OBSTRUCTIVE PULMONARY DISEASE
"blue bloater vs. pink puffer"
emPhysema has letter P (and not B) so Pink Puffer.
chronic Bronchitis has letter B (and not P) so Blue Bloater.
CIRCULATION CHECKS
"5 P's"
Pain
Paresthesia
Paralysis
Pulse
Pallor (Paleness)
CIRCULATORY CHECKS
“The 5 P’s”
Pain
Paresthesia (numbness/tingling)
Paralysis
Pulse
Pallor (Paleness)
CLEFT LIP – POST-OP NURSING CARE PLAN
"CLEFT LIP"
Crying, minimize
Logan bow
Elbow restraints
Feed with Brecht feeder
Teach feeding techniques; two months of age (average age at repair)
Liquid (sterile water), rinse after feeding
Impaired feeding (no sucking)
Position (never on abdomen)
CLEFT LIP POST OP CARE
"CLEFT LIP"
Choking
Lie on back
Evaluate Airway
Feed Slowly
Teaching
Larger nipple opening
Incidence increase in males
Prevent crust formation and aspiration
CLIENT IN TRACTION NURSING CARE
"TRACTION"
Temperature (Extremity, Infection)
Ropes hang freely
Alignment
Circulation Check (5 P's)
Type & Location of fracture
Increase fluid intake
Overhead trapeze
No weights on bed or floor
COAGULATION TESTS
"PiTT" (PTT - I for Intrinsic pathway) - PiTTsburgh
"PeT" (PT - E for Extrinsic pathway)
**Bleeding Time – provides assessment of platelet count & function (normal value: 2-8 minutes).
**Prothrombin Time (PT) – measures effectiveness of “E”xtrinsic pathway. Mnemonic – PET (normal value: 10-15 seconds).
**Partial Thromboplastin Time (PTT) – measures effectiveness of “I”ntrinsic pathway. Mnemonic – PITT (normal value: 25-40 seconds).
**Thrombin Time (TT) – time for Thrombin to convert Fibrinogen → Fibrin (a measure of Fibrinolytic Pathway). (normal value 9-13 seconds).
COGNITIVE DISORDERS – ASSESSMENT OF DIFFICULTIES
"JOCAM"
Judgment
Orientation
Confabulation
Affect
Memory
COMA – CAUSES
"A-E-I-O-U TIPS"
Alcohol, acidosis (hyperglycemic coma)
Epilepsy (also electrolyte abnormality, endocrine problem)
Insulin (hypoglycemic shock)
Overdose (or poisoning)
Uremia and other renal problems
Trauma; temp abnormalities (hypothermia, heat stroke)
Infection (e.g., meningitis)
Psychogenic ("hysterical coma")
Stroke or space-occupying lesions in cranium
COMMON CAUSES OF POST OP FEVER
"Five W's"
Wind (pneumonia)
Water (dehydration)
Wound (infection, dehiscence)
Walking (PE)
Wonder drug (wrong antibiotic)
COMPLEMENT: FUNCTION OF C3A vs C3B
C3A: Activates Acute [inflammation].
C3B: Bonds Bacteria [to macrophages- easier digestion].
• If wish to know more than just C3:
C3a, C4a, C5a – Activate Acute.
C3b, C4b – Bind Bacteria.
CONGESTIVE HEART FAILURE
“DAM DDOG”
Digoxin
Aminophylline
Morphine
Dopamine
Diuretics
O2
Gasses - monitor (ABG)
CONGESTIVE HEART FAILURE – TREATMENT
“UNLOAD FAST”
U-upright Position
N-nitrates (in low dose)
L-Lasix
O-oxygen
A-aminophylline
D-digoxin
F-fluids (decrease)
A-afterload (decrease)
S-sodium restriction
T-test (Dig level, ABGs, K level)
COPD 4 TYPES AND HALLMARK SIGN
"ABCDE"
Asthma
Bronchiectasis
Chronic bronchitis
Dyspnea [hallmark of group]
Emphysema
CORONARY ARTERIES
Right = Right
C=Coronary
A=Artery
M=Marginal Artery
P=Posterior Interventricular Artery
Left=Left
A=Anterior Interventricular Artery
C=Circumflex Artery
CORONARY ARTERIES - LOCATION
“I have a RIGHT to CAMP if you LEFT off the AC”
Right
C = coronary
A = artery
M = marginal artery
P = posterior intraventricular artery
Left
A = anterior ventricular artery
C = circumflex artery
COR PULMONALE – SIGNS
"PleaseRead His Text"
Peripheral edema
Raised JVP
Hepatomegaly
Tricuspid incompetence
COR PULMONALE - SIGNS & SYMPTOMS
Cor pulmonale is an enlargement of the right side of the heart caused by a problem with the lungs or pulmonary vessels.
“Please Read His Text”
Peripheral edema
Raised JVP
Hepatomegaly
Tricuspid incompetence
CORTICOSTEROID ADVERSE SIDE EFFECTS
"CUSHINGS BAD MD"
Cataracts
Up all night (sleep disturbances)
Suppression of HPA axis
Hypertension/ buffalo Hump
Infections
Necrosis (avascular)
Gain weight
Striae
Bone loss (osteoporosis)
Acne
Diabetes
Myopathy, moon faces
Depression and emotional changes
CORTICOSTEROID SIDE EFFECTS
"5 S's"
Sick- easier to get sick
Sad-causes depression
Sex-increases libido
Salt-retains more and causes weight gain
Sugar-raises blood sugar
CRANIAL NERVE MNEMONICS
“Oh OhOh To Try And Fit A Gold Velvet So Heavenly”
“Oh OhOh To Touch And Feel A Girl's Vagina, Ah Heaven”
“Olympic Opium Occupies Troubled Triathletes AfterFinishing Vegas Gambling Vacations Still High”
“Old Operators Occasionally Troubleshoot Tricky Abducted Family Veterans Galloping Valiantly Across History”
“Ohohoh to touch and feel very good velvet…ah”
“Oh OhOh To Touch And Feel Very Generous Vegas Slot Handles”
“On Old Olympus Towering Top A Fine Agile German Vaulted And Hopped”
“On Old Olympus' Towering Tops, A Finn And German Viewed Some Hops!”
“On Occasion, Our Trusty Truck Acts Funny - Very Good Vehicle AnyHow”
“Oh Oh Oh To Touch And Feel A Guy's Viagra Sent ("Spinal Access") Hard-on”
“Oh Once One Takes The Anatomy Final - Very Good Vacations Are Heavenly”
“Old Opie Occasionally Tries Trigonometry and Feels Very Gloomy Vague and Hypoactive”
“OhOhOh To Tough And Feel Virgin Girls Vaginas And Hymens”
CRANIAL NERVES – SENSORY, MOTOR OR BOTH
“Some Say Marry Money But My Brother Says Big Boobs Matter More”
“Some Say Money Matters But My Brother Says Big Brains Matter More”
CRISIS SITUATIONS
“RAPE”
R- Ruthless
A- Abusive
P- Personal
E- Experience
CUSHING'S SYNDROME – SYMPTOMS
“3 S's”
Sugar (hyperglycemia)
Salt (hypernatremia)
Sex (excess androgens)
CUSHINGS VS ADDISONS
Cushing is gushing cortisol.
Addison's patient's cortisol doesn't add up.
CUSHING SYNDROME
"CUSHING"
Central obesity/Comedones (acne)
Urinary glucose increase
Striae/ Suppressed immunity
Hypercortisolism/Hypertension/Hyperglycemia/Hirsutism
Iatrogenic (Increased administration of corticosteroids)
Noniatrogenic (Neoplasms)
Glucose intolerance/ Growth retardation
"Cut C4, breathe no more"
The 3rd, 4th and 5th cervical spinal nerves innervate the diaphragm.
CYANOTIC DEFECTS
"3 T's"
Tetralogy of Fallot
Truncus Arteriosus
Tricuspid Atresia
CYCLOSPORINE – SIDE EFFECTS
“5 H's”
Hirsutism
Hyperplasia of gums
Harm to Kidneys
Hypertension
Hyperglycemia
DELEGATION - WHAT NEVER DELEGATE TO UAP
Don’t share your “TEAACUP”
T – Teach
E – Education
A – Assessment
A –
C –
U –
P – Plan
DEEP VEIN THROMBOSIS DIAGNOSIS
"DVT"
Dilated superficial veins/discoloration
Venography is gold standard
Tenderness of thigh and calf
DEHYDRATION – SEVERE
“BRAT” Diet
B- Banana
R- Rice
A- Apple
T- Toasted Bread
DEMENTIA (SENILE) – ASSESSING CHANGES
“JAMCO”
J- Judgment
A- Affect
M- Memory
C- Cognition
O- Orientation
DEMENTIA
Make sure they don't have problems with:
D-Drug and alcohol
E-Eyes and ears
M-Metabolic and endocrine disorders
E-Emotional disorders
N-Neurologic disorders
T-Tumors and trauma
I-Infection
A-Arteriovascular disease
DEPRESSION ASSESSMENT
“SIG”
S-Sleep Disturbances
I-Interest Decreased
G-Guilty Feelings
DIABETES – SIGN & SYMPTOMS
“3 P's”
Polydipsia (very thirsty)
Polyphagia (very hungry)
Polyuria (urinary frequency)
DIABETES MELLITUS - TYPE 1
“3 P's” (Signs & Symptoms)
P-Polyuria(excessive urination)
P- Polydipsia (excessive thirst)
P- Polyphagia (excessive hunger)
DIABETIC FITNESS
“FIT” exercise guide
F - Frequency (3x per week)
I - Intensity (60-80% of Maximal Heart Rate)
T- Time (Aerobic Activity)
DIABETIC KETOACIDOSIS – MANAGEMENT
“KING UFC”
K+ (potassium)
Insulin (5u/hr. Note: sliding scale not recommended in UK)
Nasogastric tube (if patient comatose)
Glucose (once serum levels drop to 12)
Urea (check it)
Fluids (crystalloids)
Creatinine (check it)/ Catheterize
DIABETES COMPLICATIONS
"KNIVES”
Kidney
Neuropathy
Infection
Vascular (Cardio)
Eyes
Skin lesions
DIABETES TYPE 1 SYMPTOMS
"3 P's"
Polyuria (excessive urination)
Polydipsia (excessive thirst)
Polyphagia (excessive hunger)
DIABETIC KETOACIDOSIS (DKA) - CAUSES
"5 I's"
Infection
Ischemia (cardiac, mesenteric)
Infarction
Ignorance (poor control)
Intoxication (alcohol)
DIABETIC KETOACIDOSIS (DKA) - SIGNS
"DKA"
Dehydrated
Ketones/Kussmaul breathing/K drops
Acidosis
DIALYSIS – WHO NEEDS DIALYSIS?
“AEIOU” (check the vowels)
A- Acid-Base Problems
E- Electrolyte Problems
I- Intoxications
O- Overload of fluids
U - Uremic Symptoms
DIALYSIS CANDIDATES
"AEIOU"
Acid-Base Problems
Electrolyte Problems
Intoxications
Overload of fluids
Uremic Symptoms
DIARRHEA - TREATMENT
The BRAT diet is a bland diet that is low in protein, fiber, and fat. It is thought to be easy on the GI tract and helpful to lesson diarrhea. It is not used commonly anymore because of the lack of protein in the diet.
“BRAT”
Banana
Rice
Apple
Toast
DIC
“PCFCFMCF”
Platelets
Clotting Factors Consumed,
Form Microthrombi,
Coagulation Fails
DIET FOR SEVERE DEHYDRATION
"BRAT"
Banana
Rice
Apple
Toasted Bread
DILATED PUPILS
"FEAR, FITS OR FAST LIVING”
Fear: panic, extreme anxiety
Fits: seizures
Fast Living: cocaine, crack, phencyclidine (PCP)
DISSEMINATED INTRAVASCULAR COAGULATION DIC
“D-I-S-S-E-M-I-N-A-T-E-D”
D-Dx: D dimer
I-Immune complexes
S-Snakebite, shock, heatstroke
S-SLE
E-Eclampsia, HELLP syndrome
M-Massive tissue damage
I-Infections: viral and bacterial
N-Neoplasms
A-Acute promyelocytic leukemia
T-Tumor products: Tissue Factor (TF) and
TF-like factors released by carcinomas of pancreas, prostate, lung, colon, stomach
E-Endotoxins (bacterial)
D-Dead fetus (retained)
DIURETICS
"Leak Over The CAN"
Loop diuretics
Osmotics
Thiazides
Carbonic anhydrase inhibitors
Aldosterone inhibitors
Na (sodium) channel blockers
DIGOXIN – TOXICITY & GI SYMPTOMS
“VANDA”
Vomiting
Anorexia
Nausea
Diarrhea
Abdominal pain
DRUGS THAT INTERACT
"These Drugs Can Interact"
Theophylline
Dilantin
Coumadin
losone (Erythromycin)
DRUG INTERACTIONS
Coumadin and ilosone: ilosone can increase the effects of Coumadin, increased risk for bleeding
Coumadin and Dilantin: potential for increased effects of both Coumadin and Dilantin
Theophylline and Dilantin: if taken orally they can interfere with absorption of each other and decrease medication effect
“TDCI (These Drugs Can Interact)”
Theophylline
Dilantin
Coumadin
Ilosone (Erythromycin)
DYSPNEA
"6 P's"
Pulmonary Bronchial Constriction
Possible Foreign Body
Pulmonary Embolus
Pneumothorax
Pump Failure
Pneumonia
DYSPNEA
Pulmonary bronchial constriction prevents passage of air into lungs which contributes to dyspnea. (Possible) foreign bodies can become trapped or logged within trachea restricting air flow. PEs can prevent complete oxygenation of the blood in alveoli d/t restricted blood flow. Pneumothorax collapses the lung and prevents full expansion, restricting oxygenation. Pump failure refers to heart not beating appropriately. If heart is not perfusing the lungs than lungs are unable to oxygenate blood. Pneumonia leads to poor lung ventilation. These are six major causes for dyspnea.
“The 6 P’s”
Pulmonary Bronchial Constriction
Possible Foreign Body
Pulmonary Embolus (PE)
Pneumothorax
Pump Failure
Pneumonia
DYSPNEA DIFFERENTIAL
"3-A's, 3-P's, 3-C's, 3-M's"
“3-A's” Three Airways:
Airway obstruction
Anaphylaxis
Asthma
“3-P's” Three Pulmonary's:
Pneumothorax
PE
Pulmonary edema
“3-C's” Three Cardiacs:
Cardiogenic pulmonary edema
Cardiac ischemia
Cardiac tamponade
“3-M's” Three Metabolics:
(DOC) DKA
Organophosphates (pesticides)
Carbon monoxide poisoning
E. COLI – CAUSES
“DUNG”
Diarrhea
UTI
Neonatal meningitis
Gram negative sepsis
ECG LEAD PLACEMENT
White - right
Clouds over grass (white over green)
Smoke over fire (black over red)
Chocolate on the stomach (brown)
ELECTROLYTES
“PISO”
P- Potassium
I- Inside
S- Sodium
O- Outside
ELECTROLYTES - LOCATION IN CELLS
Potassium and sodium are the two most abundant cations in the body and have an inverse relationship in regards to intracellular and extracellular concentrations. Potassium is primarily located within the cell and sodium is primarily located outside the cell.
“PISO”
Potassium
Inside the cell
Sodium
Outside the cell
EMERGENCY DRUGS
The above drugs work in a variety of emergency settings. Lidocaine can be used in emergency situations for ventricular arrhythmias. Epinephrine is a vital drug in the ACLS protocol. Atropine can be given with symptomatic bradycardia. Narcan is a reversal agent for opiate overdose.
“Drugs to LEAN on”
Lidocaine
Epinephrine
Atropine Sulfate
Narcan
EMERGENCY DRUGS TO “LEAN” ON
L- Lidocaine
E - Epinephrine
A- Atropine Sulfate
N - Narcan
EMERGENCY TRAUMA ASSESSMENT
“ABCDEFGHI”
A-airway
B-breathing
C-circulation
D-disability
E-examine
F-Fahrenheit
G-get Vitals
H-head to Toe Assessment
I-intervention
EMERGENCY MEDS
"drugs to LEAN on"
Lidocaine
Epinephrine
Atropine Sulfate
Narcan
EMERGENCY MI TREATMENT
"MONATAS"
Morphine
Oxygen
Nitroglycerine
ASA
Thrombolytics
Anti-Coagulants
Stool Softeners
EMERGENCY TRAUMA GENERAL PROCEDURE
"ABCDEFGHI"
Airway
Breathing
Circulation
Disability
Examine
Fahrenheit
Get Vitals
Head to Toe Assessment
Intervention
ENDOCARDITIS
“FROM JANE”
Fever
Roth's Spots
Osler's nodes
Murmur
Janeway Lesions
Anemia
Nail Hemorrhage (splinter hemorrhage)
Emboli
ENERGY DECREASED
“CAPS”
C-concentration decreased
A-appetite
P-psychomotor function decreased
S-suicidal Ideations
EPIGLOTTITIS
“AIR RAID”
A-airway Closed
I-increased Pulse
R-restlessness
R-retractions
A-anxiety Increased
I-inspiratory Stridor
D-drooling
EPIGLOTTITIS – SIGNS & SYMPTOMS
“ADD AIR NURSE”
A-abnormal posturing (tripod)
D-dysphagia → D-drooling
D-different speaking (muffled/soft)
A-apprehension
I-increased temperature (high)
R-rapid onset
N-nasal flaring
U-using accessory muscles
R-retractions (chest)
S-stridor (inspiratory)
E-enlarged epiglottis (enlarged on x-ray or visible)
**cough NOT typically present**
EPIGLOTTITIS - ASSOCIATED S/S
The epiglottis is a flap of cartilage that covers the entrance to our airway when we swallow food. Inflammation of the epiglottis can close off the airway entirely.
“AIR RAID”
Airway Closed
Increased Pulse
Restlessness
Retractions- occur when the muscles between the ribs pull inward
Anxiety Increased
Inspiratory Stridor- high-pitched breath sound resulting from turbulent airflow in the larynx
Drooling
EPISIOTOMY ASSESSMENT
"REEDA"
Redness
Edema
Ecchymosis
Discharge, Drainage
Approximation
EPISIOTOMY ASSESSMENT
"REEDA"
Redness
Edema
Ecchymosis
Discharge
Approximation of skin
EPISIOTOMY HEALING – EVALUATION OF
“REEDA”
R- Redness
E- Edema
E - Ecchymosis
D - Discharge, Drainage
A – Approximation
EYE MEDICATIONS
Mydriatic = dilated pupils
Miotic = tiny (constricted) pupils
FETAL WELL BEING DIAGNOSTICS
"ALONE"
Amniocentesis
L/S Ratio
Oxytocin test
Non-Stress test
Estriol level
FETAL WELL-BEING – ASSESSMENT TESTS
“ALONE”
A- Amniocentesis
L- L/S Ratio
O- Oxytocin Test
N- Non-Stress Test
E- Estriol Level
FOODS TO AVOID WITH CELIAC DISEASE
"BROW"
Barley
Rye
Oats
Wheat
FOUR CAUSES OF CELL INJURY
"TIPD the scale of homeostasis"
Toxin or other lethal (cytotoxic) substance
Infection
Physical insult or injury
Deficit or lack of water, oxygen, or nutrients
FRACTURE
“PRICE”
Pressure
Rest
Ice
Compression
Elevation
GLUTEN FREE DIET
“ROW”
R- Rye
O- Oats
W- Wheat
GLUTEN FREE DIET
Gluten is a protein found in wheat, barley and rye. People with gluten allergies can be affected by even trace amount of gluten in foods. Oats do not contain gluten, but they are often milled in the same factories as wheat. Always check food labels to make sure a product is gluten free.
“BROW“
Barley
Rye
Oats
Wheat
GOUT TRIGGERS
"DARK"
Diuretics
Alcohol
Renal disease
Kicked (trauma)
And, the attack occurs most often at night [thus "dark"]
HEART FAILURE – LEFT-SIDED
“CHOP”
C- Cough
H- Hemoptysis
O- Orthopnea
P- Pulmonary Congestion (crackles/ rales)
HEART FAILURE - LEFT-SIDED
Left sided heart failure will primarily manifest as pulmonary edema as the left side of the heart moves blood from the lungs throughout the body. If this portion of the pump is broken the blood will become "backed up" within the lungs. Left sided heart failure can often lead to right sided heart failure.
“CHOP”
Cough
Hemoptysis
Orthopnea
Pulmonary Congestion (crackles/rales)
HEART FAILURE – RIGHT SIDED
“HEAD”
H- Hepatomegaly
E- Edema (Bipedal)
A- Ascites
D- Distended Neck Vein
HEART FAILURE - RIGHT-SIDED
Right sided heart failure will primarily manifest as central edema as the right side of the heart takes fluid from the body and moves it forward. If this portion of the pump is broken, fluid will become "backed up" behind the pump and therefore within the body.
“HEAD”
Hepatomegaly
Edema (Bipedal)
Ascites
Distended Neck Vein
HEART VALVE LOCATION – RT TO LT
"A Permanently Temperamental Man"
Aortic
Pulmonary
Tricuspid
Mitral
HEART AUSCULTATION ORDER
"APETM"
Atrial
Pulmonic
Erb's point
Tricuspid
Mitral
HEART SOUNDS - 1
Aortic and Pulmonic (A&P): 2 words, 2 spaces; these coincide in that they are both in the 2nd intercostal space. A is immediately to the right of the sternum; P is immediately to the left of the sternum.
Erb's Point: Erb has 3 letters; you can find this point in the 3rd intercostal space (just to the left of the sternum), also the 3rd space to auscultate
Tricuspid: 5th intercostal, just to the left of the sternum. Tri = 3, this is the fourth place to auscultate in the 5th intercostal space!
Mitral: Mit sounds similar to mid…Midclavicular area and straight down just below nipple line
“APE To Man”
“All People Enjoy Time Magazine”
“Apple Pie Especially Tastes Mmmm”
Aortic
Pulmonic
Erbs points
Tricuspid
Mitral
HEMOLYTIC ANEMIA TYPES
“SHEEP TiT”
Sickle cell
Hereditary splenocytosis
Enzyme deficiencies: [G6P, pyruvate kinase]
Erythroblastosis fetalis
Paroxysmal nocturnal hemoglobinuria
Trauma to RBCs
Immunohemolytics: [warm Ab, cold Ag]
Thalassemia’s: [alpha, beta]
HEMOPHILIA: TYPE A FACTOR
Hemophilia A: problems with VIII factor
(number V as an inverted A).
HEPATITIS TRANSMISSION ROUTES
"Vowels are bowels"
Hepatitis A and E transmitted by fecal oralroute
HISTAMINE: FEATURES
“HISTAMINE”
HCL production
Inflammation
Strong vasodilator
Therapeutic value none
Allergy
Mast cells
Neurotransmitter/ Narrow airways
IgE
HOLISTIC CARE COMPONENETS
“SPISE”
S - Spiritual
P - Physical
I - Intellectual
S - Social
E - Emotional
HORMONES – THAT ↑ BLOOD GLUCOSE
“STENGG”
Somatotropin (growth hormone)
Thyroid Hormones (thyroxine and triiodothyronine)
Epinephrine
Norepinephrine
Glucagon
Glucocorticosteroids
HYPERCALCEMIA – S/S RHYME
“Groans, Moans, Bones, Stones, Overtones”
Groans: constipation
Moans: pain - joint aches
Bones: calcium loss from bones, bone metastasis
Stones: kidney stones
Overtones: psychiatric overtones - depression, confusion
HYPERCALCEMIA
"MD PIMPS ME"
Malignancy
Diuretics (thiazide the main culprit)
Parathyroid (hyperparathyroidism)
Immobilization/ Idiopathic
Megadoses of vitamins A,D
Paget's disease
Sarcoidosis
Milk alkali syndrome
Endocrine (Addison's disease, thyrotoxicosis)
HYPERGLYCEMIA – SIGNS
“3 P's”
Polydipsia
Polyphagia
Polyuria
Hot and dry, sugar high
HYPERGLYCEMIA/HYPOGLYCEMIA
During hyperglycemia, a patient is dehydrated with elevated blood sugars. In hypoglycemia, a patient is cold and clammy and need a carbohydrate source to bring their sugars back up to a normal range.
Hyper - hot/dry = sugar high
Hypo- cold/clammy = needs candy
HYPERGLYCEMIA - SIGNS AND SYMPTOMS
“Groans-Moans-Bones-Stones-Overtones”
Groans - constipation
Moans - joint pain
Bones - loss of calcium from bones
Stones - kidney stones
Overtones - psychiatric overtones (confusion, depression)
HYPERKALEMIA
“MURDER & SALT"
Signs & Symptoms
“MURDER”
M - Muscle weakness
U - Urine, oliguria, anuria
R - Respiratory distress
D - Decreased cardiac contractility
E - ECG changes
R - Reflexes, hyperreflexia, or areflexia (flaccid)
Causes
“MACHINE”
M - Medications - ACE inhibitors, NSAIDS
A - Acidosis - Metabolic and respiratory
C - Cellular destruction - Burns, traumatic injury
H - Hypoaldosteronism/ hemolysis
I - Intake - Excessive
N - Nephrons, renal failure
E - Excretion - Impaired
HYPERKALEMIA – SIGNS
“MURDER”
Muscle Weakness
Urine (oliguria or anuria)
Respiratory Distress
Decreased cardiac contractility
EKG Changes (Peaked T waves or small P waves)
Reflexes (hyper or hypo)
HYPERKALEMIA – MANAGEMENT
“KIND”
K- Kayexalate (orally/ enema)
I- Insulin
N- Na HCO3
D- Diuretics (Furosemide & Thiazides)
HYPERKALEMIA CAUSES
"MACHINE"
Medications - ACE inhibitors, NSAIDS
Acidosis - Metabolic and respiratory
Cellular destruction - Burns, traumatic injury
Hypoaldosteronism, hemolysis
Intake - Excessive
Nephrons, renal failure
Excretion – Impaired
HYPERKALEMIA - CAUSES
“the hyperkalemia MACHINE”
Medications - ACE Inhibitors, NSAIDS, potassium-sparing diuretics
Acidosis - Metabolic and respiratory
Cellular destruction - burns, traumatic injury, hemolysis
Hypoaldosteronism - Addison's
Intake- excessive
Nephrons- renal failure
Excretion - Impaired
HYPERKALEMIA - SIGNS AND SYMPTOMS
“MURDER”
Muscle weakness
Urine- oliguria, anuria
Respiratory distress
Decreased cardiac contractility
ECG changes
Reflexes- hyperreflexia or areflexia (flaccid)
HYPERKALEMIA - MANAGEMENT
“AIRED”
Administer IV Calcium - to immediately decrease cardiac toxicity Increase excretion - via both stool (kayexlate) and urine (diuretics) Remove sources of potassium - from all sources, including enteral, parenteral, IV, and PO
Enhance potassium uptake into cells - insulin, glucose, sodium bicarb, beta-adrenergic antagonists
Dialysis - emergent response for patients with lethal hyperkalemia
HYPERKALEMIA - DEFINITION
Hyperkalemia is elevated potassium in the blood. Typical levels of K+ in blood are 3.7-5.2 mEq/L.
In acidosis and cellular destruction, potassium shifts from inside the cell to the blood stream. Medications and kidney damage can decrease urinary excretion of potassium. Excessive intake of potassium can also lead to hyperkalemia. Potassium is necessary for the transmission of electrical impulses in heart and skeletal muscle; therefore increased potassium can cause ECG changes.
HYPERKALEMIA SYMPTOMS
"MURDER"
Muscle weakness
Urine, oliguria, anuria
Respiratory distress
Decreased cardiac contractility
ECG changes (peaked T at 6, prolonged PR at 7, absent P andwide QRS at 8-9)
Reflexes, hyperreflexia, or areflexia (flaccid)
HYPERKALEMIA TREATMENT
"KIND"
Kayexalate (orally/ enema)
Insulin
Na HCO3
Diuretics (Furosemide & Thiazides)
HYPERNATREMIA - CAUSES
“MODEL”
Medications / Meals
Osmotic diuretics
Diabetes insipidus
Excessive water loss
Low water intake
HYPERNATREMIA – SIGNS
“FRIED”
Fever
Restless
Increased BP
Edema
Decreased Urinary Output
HYPERNATREMIA – FRIED SALT
“FRIED”
F - Fever (low), flushed skin
R - Restless (irritable)
I - Increased fluid retention & increased BP
E - Edema (peripheral and pitting)
D - Decreased urinary output, dry mouth
“SALT”
S - Skin flushed
A - Agitation
L - Low-grade fever
T - Thirst
CAUSE: “MODEL”
Medications, meals
Osmotic diuretics
Diabetes insipidus
Excessive water loss
Low water intake
HYPERNATREMIA CAUSES
"6 D's": Diuretics
Dehydration
Diabetes insipidus
Docs (iatrogenic)
Diarrhea
Disease: kidney, sickle cell, etc.
HYPERNATREMIA SYMPTOMS
"FRIED"
Fever (low grade), flushed skin
Restless (irritable)
Increased fluid retention and increased BP
Edema (peripheral and pitting)
Decreased urinary output, dry mouth
HYPERNATREMIA - SIGNS AND SYMPTOMS
“You are FRIED from learning about sodium”
Flushed skin
Restlessness: irritability
Increased fluid retention and increased BP
Edema: peripheral and pitting
Deceased urinary output, dry mouth
HYPERNATREMIA - SIGNS AND SYMPTOMS
“SWINE” (bacon has high amounts of sodium)”
Sleepy - lethargy (typically the very first sign!)
Weakness
Irritability
Neuromuscular excitability
Edema
HYPERNATREMIA - SIGNS AND SYMPTOMS
“SALT”
Skin flushed
Agitation
Low grade fever
Thirst
HYPERSENSITIVITY REACTIONS
“ACID”
Type 1 - Anaphylactic
Type 2 - Cytotoxic
Type 3 - Immune Complex
Type 4 - Delayed Hypersensitivity
HYPERSENSITIVITY REACTIONS
Gell and Goombs nomenclature
“ACID” From I to IV:
Anaphylactic type: type I
Cytotoxic type: type II
Immune complex disease: type III
Delayed hypersensitivity cell mediated: type IV
HYPERTENSION – COMPLICATIONS
“4 C's”
C- Coronary ArteryDisease
C- Coronary Rheumatic Fever
C- Congestive Heart
C- Cardio Vascular Accident
HYPERTENSION – COMPLICATIONS
“4 C's”
CAD (coronary artery disease)
CHF (congestive heart failure)
CRF (chronic renal failure)
CVA (cardiovascular accident; now called brain attack or stroke)
HYPERTENSION - COMPLICATIONS
“The 4 C’s”
Coronary Artery Disease
Coronary Rheumatic Fever
Congestive Heart Failure
Cerebral Vascular Accident
HYPERTENSION – NURSING CARE
“DIURETIC”
D-daily Weight
I-intake and Output (I & O)
U-urine Output
R-response of BP
E-electrolytes
T-take Pulses
I-ischemic Episodes (TIA)
C-complications: 4C's
HYPERTENSION – NURSING CARE PLAN
"I-TIRED"
Intake and output (urine)
Take blood pressure
Ischemia attack, transient (watch for TIAs)
Respiration, pulse
Electrolytes
Daily weight
HYPERTENSION NURSING CARE
"DIURETIC"
Daily Weight
Intake and Output (I & O)
Urine Output
Response of BP
Electrolytes
Take Pulses
Ischemic Episodes (TIA)
Complications: 3C's (CAD, CHF, CVA)
HYPERTENSION TREATMENT
"ABCD"
ACE inhibitors
Beta blockers
Calcium antagonists
Diuretics (sometimes vaso"D"ilators also)
HYPOCALCEMIA
“CATS”
C - Convulsions
A - Arrhythmias
T - Tetany
S - Spasms and stridor
HYPOCALCEMIA
Hypocalcemia is low levels of calcium in the blood. Calcium in the blood can be bound to proteins, bound to anions like phosphate, or ionized. Large of stores of calcium are in the bone. Calcium blocks sodium channels, inhibiting depolarization of muscle and nerve fibers. The effects of hypocalcemia are a result of muscle fibers being more excitable.
“CATS of hypocalcemia”
Convulsions
Arrhythmias
Tetany
Spasms and stridor
HYPOCALCEMIA SYMPTOMS
"CATS": Convulsions
Arrhythmias
Tetany
Spasms and stridor
HYPOGLYCEMIA – SIGNS
“TIRED”
Tachycardia
Irritability
Restlessness
Excessive Hunger
Diaphoresis
Cold and Clammy, need some candy
HYPOGLYCEMIA
“TIRED”
Tremors, tachycardia
Irritability
Restlessness
Extreme
Diaphoresis
HYPOGLYCEMIA – SIGNS &SYMPTOMS
"DIRE"
Diaphoresis
Increased pulse
Restless
Extra hungry
HYPOGLYCEMIA - SIGNS AND SYMPTOMS
During hypoglycemia the serum glucose level is too low. Cells of the body can't get adequate energy supply when there is not enough sugar in the blood. You will feel TIRED and have the symptoms listed above.
“TIRED”
Tachycardia
Irritability
Restless
Excessive Hunger
Diaphoresis / Depression
HYPOGLYCEMIA CAUSES
"RE-EXPLAIN": Renal failure
Exogenous
Pituitary
Liver failure
Alcohol
Infection
Neoplasm (insulinoma)
HYPOGLYCEMIA VS HYPERGLYCEMIA
"Hot and Dry"
"Cold and Clammy": Hot and dry = sugar high
Cold and clammy = need some candy
HYPOKALEMIA – SIGNS
“6 L's”
Lethargy
Lethal cardiac arrhythmia
Leg cramps
Limp muscles
Low, shallow respirations
Less Stool (constipation)
HYPOKALEMIA
SKELETAL MUSCLE WEAKNESS
U-WAVE ON ECG
CONSTIPATION
TOXICITY TO DIGOXIN
IRREGULAR WEAK PULSE
OTOSTASIS
NUMBNESS PARESTHESIA
Signs and Symptoms
“6 L's”
Lethargy
Leg cramps
Limp muscles
Low, shallow respirations
Lethal cardiac dysrhythmias
Lots of urine (polyuria)
HYPOKALEMIA - SIGNS AND SYMPTOMS
“6 L’s”
Lethargy
Leg cramps
Limp muscles
Low, shallow respirations
Lethal cardiac dysrhythmias
Lots of urine (polyuria)
HYPONATREMIA SYMPTOMS
"SALT LOSS"
Stupor/coma
Anorexia, N&V
Lethargy
Tendon reflexes decreased
Limp muscles (weakness)
Orthostatic hypotension
Seizures/headache
Stomach cramping
HYPONATREMIA – SIGNS & SYMOTOMS
“SALT LOSS”
S-stupor/coma
A-anorexia, N&V
L-lethargy
T-tendon reflexes decreased
L-limp muscles (weakness)
O-orthostatic hypotension
S-seizures/headache
S-stomach cramping
HYPONATREMIA – SIGNS & SYMPTOMS
“SALT”
Stupor/coma
Anorexia
Lethargy
Tendon reflexes decreased
“LOSS”
Limp muscles
Orthostatic hypotension
Seizures/headache
Stomach cramps
HYPONATREMIA - S/S
Hyponatremia is decreased levels of sodium in the blood. It can be caused by inadequate sodium or excess free water which leads to lower concentration of sodium. Sodium and potassium work together to allow depolarization of muscles. Low sodium levels can limit this ability and cause muscle weakness. Sodium in the blood helps maintain the oncotic pressure. If fluid leaves the blood vessels, it can lead to decreased blood pressure.
“SALT LOSS”
Stupor/coma
Anorexia, N&V
Lethargy
Tendon Reflexes decreased
Limp muscles (weakness)
Orthostatic hypotension
Seizures/headache
Stomach cramping
HYPOXIA – EARLY SIGNS
“RAT”
Restlessness
Agitation
Tachycardia
HYPOXIA - SIGNS AND SYMPTOMS
Patients experiencing hypoxia will initially demonstrate signs of anxiety and restlessness. As hypoxia continues, the patient’scondition deteriorates to bradycardia and dyspnea.
“RAT BED”
Early Hypoxia:
Restlessness
Anxiety
Tachycardia / Tachypnea
Late Hypoxia:
Bradycardia
Extreme Restlessness
Dyspnea
HYPOXIA – LATE SIGNS
Bradycardia
Extreme restlessness
Dyspnea
Cyanosis
HYPOXIA – SYMPTOMS IN PEDIATRICS
“FINES”
F-feedingdifficulty
I-inspiratory Stridor
N-nares Flares
E-expiratory Grunting
S-sternal Retractions
IMMUNITY – PASSIVE VS. ACTIVE
"Pay for Passive, Active Ages"
Passive: Pay for a shot of antibodies for fast results following exposure to Rabies, etc.
Active: Slow onset "aging" and memory.
IMMUNOGLOBULINS
"MADGE"
IgM
IgA
IgD
IgG
IgE
INCREASE ICP - CUSHINGS TRIAD
“HYPER-BRADY-BRADY”
Hypertension (wide pulse pressure)
Bradycardia
Bradypnea
INFLAMMATION – SIGNS
“PRISH”
Pain
Redness
Immobility (loss of function)
Swelling
Heat
INFLAMMATION
“HIPER”
H-heat
I-induration
P-pain
E-edema
R-redness
INFLAMMATION
"HIPER"
Heat
Induration
Pain
Edema
Redness
INFLAMMATION - SIGNS AND SYMPTOMS
In response to tissue damage the body elicits the inflammatory response in attempt to rid itself of the initial offender. These attempts can sometimes become more of a problem than a solution.
“HIPER”
Heat
Indurations (hardening)
Pain
Edema
Redness
INSULIN MIXING
"Not Ready, Ready Now"
Air into NPH
Air into Regular
Draw up Regular
Draw up NPH
IUD – POTENTIAL PROBLEMS WITH USE
"PAINS"
Period (menstrual: late, spotting, bleeding)
Abdominal pain, dyspareunia
Infection (abnormal vaginal discharge)
Not feeling well, fever or chills
String missing
KETOACIDOSIS CAUSES
"SAD"
Starvation
Alcohol
DM
K+ INCREASING DRUGS
"K BANK"
K-sparing diuretics
Beta blockers
ACE inhibitors
NSAIDs
K supplements
KEY QUESTIONS NEEDED IN AN EMERGENCY HISTORY TAKING SITUATION
"AMPLE"
Allergies
Medication
Past medical history
Last meal
Events and environment related to injury
LACTATION HORMONES
"Prolactin vs Oxytocin"
PRO-lactin stimulates mammaryglands to PRO-duce milk.
Oxytocin stimulates mammary glands to Ooze (release)milk.
LEUKEMIA SYMPTOMS
"ANT"
Anemia
Neutropenia
Thrombocytopenia
LEUKEMIA – SYMPTOMS
“ANT”
A- Anemia
N- Neutropenia
T- Thrombocytopenia
LEUKEMIA - SIGNS AND SYMPTOMS
Leukemia can lead to anemia as it will disrupt the production of blood within the bone marrow. Neutropenia is a natural result of leukemia as the body's supply of white blood cells is decreased. Thrombocytopenia is a reduced platelet count which can also result from leukemia, especially in infants.
“ANT”
Anemia
Neutropenia
Thrombocytopenia
LEUKOCYTES: GRANULATED AND AGRANULATED
"BEN Loves Money"
Granulocytes:
B-basophil
E-eosinophil
N-neutrophil
•Agranulocytes:
L-lymphocytes
M-monocytes
Alternatively: Granpa BEN..."to keep the granulated group straight.
LIDOCAINE TOXICITY
“SAMS”
S-slurred Speech
A-altered Central Nervous System
M-muscle Twitching
S-seizures
LIDOCAINE TOXICITY - SIGNS AND SYMPTOMS
Lidocaine is an anesthetic that prevents painful impulses from reaching the brain.
“SAMS”
Slurred Speech
Altered Central Nervous System
Muscle Twitching
Seizures
LIVER FUNCTIONS
“PUSH DoG”
Protein Synthesis
Urea synthesis
Storage
Hormone synthesis
Detoxification
Glucose and fat metabolism
LOW CHOLESTEROL DIET
“3 C's” to avoid…
Cake
Cookies
Cream (dairy, e.g., milk, ice cream)
LUPUS SIGNS AND SYMPTOMS
"SOAP BRAIN"
Serositis [pleuritis, pericarditis]
Oral ulcers
Arthritis
Photosensitivity
Blood [all are low - anemia,
Leukopenia, thrombocytopenia]
Renal [protein]
ANA
Immunologic [DS DNA, etc.]
Neurologic [psych, seizures]
MACROCYTOSIS: NON-B12/FOLATE CAUSES
“ALPHA NERD”
Alcohol
Liver disease
Pregnancy
Hemolysis (especially chronic)
Agglutination
Neoplasia (including myelodysplasia)
Endocrine (hypothyroidism)
Reticulocytes
Drugs (especially myelosuppressives like chemotherapy, anti-HIV meds)
MALIGNANCIES THAT METASTISIZE TO BONE
"Laging Panalo Kung Taga Bulacan"
Lung
Prostate
Kidney
Thyroid
Breast
MALIGNANT HYPERTHERMIA – TREATMENT
"Some Hot Dude Better Give Iced Fluids Fast"
Hot dude = hypothermia
Stop triggering agents
Hyperventilate/ Hundred percent oxygen
Dantrolene (2.5mg/kg)
Bicarbonate
Glucose and insulin
IV Fluids and cooling blanket
Fluid output monitoring/ Furosemide/ Fast heart [tachycardia]
MANIC ATTACK – MAJOR SYMPTOMS
“DIG FAST”
D-Distractibility
I- Indiscretion
G- Grandiosity
F- Flight of Ideas
A- Activity Increase
S- Sleep Deficit
T- Talkative
MANIPULATION – NURSING PLAN
"3C's"
Cooperation
Compromise
Collaboration
MED ADMINISTRATION
"DR TMP"
Dose
Route
Time
Patient
Med
MED ADMIN – 5-CHECKS
“TRAMP”
T = right TIME
R = right ROUTE
A = right AMOUNT
M = right MEDICATION
P = right PERSON
MELANOMA – CHARACTERISTICS
“ABCD”
Asymmetrical
Borders irregular
Color dark and variation
Diameter is large (>6 mm)
MENTAL RETARDATION – NURSING CARE PLAN
“3 R's"
Regularity (provide routine and structure)
Reward (positive reinforcement)
Redundancy (repeat)
METABOLIC ACIDOSIS CAUSES
"LUSK"
Lactic
Uremic
Salicylate
Ketoacidosis
MG SO4 TOXICITY
BP decrease
Urine output decrease
Respiratory rate decrease
Patellar reflex absent
MICROCYTIC ANEMIA: CAUSES
"Find Those Small Cells"
Fe deficiency
Thalassemia
Sideroblastic
Chronic disease
MINOR BLEEDING SYMPTOMS
"BEEP"
Bleeding gums
Ecchymosis (bruises)
Epistaxis (nosebleed)
Petechiae (tiny purplish spots)
MI SEQUENCE OF ELEVATED ENZYMES (FIRST TO LAST)
"Time to CALL 911"
Troponin
CK-MB
AST
LDH1
MULTIPLE MYELOMA – SYMPTOMS
“CRAB”
Calcium level elevated
Renal Failure
Anemia
Bone Breakdown and resorption
MYOCARDIAL INFARCTION (MI)
"BOOMAR" (basic management)
Bed rest
Oxygen
Opiate
Monitoring
Anticoagulation
Reduce clot size
MYOCARDIAL INFARCTION - MANAGEMENT
“MONATAS”
M-Morphine
O- Oxygen
N- Nitrates (Nitroglycerin)
A- Aspirin (ASA)
T- Thrombolytics
A- Anti-Coagulants
S- Stool Softeners
MYOCARDIAL INFARCTION – IMMEDIATE TREATMENT
“DOGASH”
Diamorphine
Oxygen
GTN spray
Aspirin 300mg
Streptokinase
Heparin
“MONA”
Morphine
Oxygen
Nitrates
Aspirin
MYOCARDIAL INFARCTION - IMMEDIATE TREATMENT
After a MI the immediate treatment is morphine sulfate, O2, nitroglycerin, and aspirin. Morphine works to decrease pain and difficulty breathing, O2 insures that poorly oxygenated tissues receive the required O2, nitroglycerin is a potent vasodilator and aids in restoring oxygenation to tissues, aspirin helps to thin the blood and increase tissue perfusion.
“MONA”
Morphine sulfate
Oxygen
Nitroglycerin
ASA (aspirin)
MYOCARDIAL INFRACTION - MANAGEMENT
“MONATAS”
Morphine
Oxygen
Nitrates (Nitroglycerin)
Aspirin (ASA)
Thrombolytics
Anticoagulants
Stool Softeners
MYOCARDIAL INFARCTION – TREATMENT
“MONA"
Monitor/Morphine
Oxygen
Nitroglycerin
Aspirin
NEPHRITIC SYNDROME
“PHARAOH”
Proteinuria and Edema
Hematuria
Azotemia
RBC casts
Anti-strep tires (if post strep)
Oliguria
Hypertension
NEPHROTIC SYNDROME – SYMPTOMS
“People Have Endless Appetites”
Proteinuria
Hyperlipidemia
Edema
Albuminuria and hypoalbuminemia
NEUROLEPTIC MALIGNANT SYNDROME
“FEVER”
Fever
Encephalopathy
Vitals unstable
Elevated enzymes (CK)
Rigidity of muscles
NEUROVASCULAR CHECK
Pain
Pulselessness
Paresthesia
Paralysis
Pallor
NEWBORN ASSESSMENT COMPONENTS
"APGAR"
Appearance
Pulse
Grimace
Activity
Respiratory effort
NINE-POINT POSTPARTUM ASSESSMENT
“BUBBLEHER”
B- Breasts
U- Uterus
B- Bladder
B- Bowel function
L- Lochia
E- Episiotomy
H- Homan's sign
E- Emotional Status
R- Respiratory System
NURSING PROCESS – ANA
“AD"PO"IE”
A- Assessment
D- Diagnosis
O- Outcome identification
P- Planning interventions
I- Implementation
E- Evaluation
OB NON-STRESS TEST
“NNN” (3 negatives in a row to interpret results of Non-Stress Test)
Non-reactive
Non-stress is
Not good
OBSTETRIC (MATERNITY) HISTORY
"GTPAL"
Gravida
Term
Preterm
Abortions (SAB, TAB)
Living children
OPIOID EFFECTS
"BAD AMERICANS"
Bradycardia & hypotension
Anorexia
Diminished pupillary size
Analgesia
Miosis
Euphoria
Respiratory depression
Increased smooth muscle activity (biliary tract constriction)
Constipation
Ameliorate cough reflex
Nausea and vomiting
Sedation
ORAL BIRTH CONTROL PILLS (BCP) – SERIOUS COMPLICATIONS
“ACHES”
A- AbdominalPain
C - Chest Pain
H - Headache
E - Eye Problems
S - Severe Leg Pain
ORAL CONTRACEPTIVES – SIGNS OF POTENTIAL PROBLEMS
"ACHES"
A-abdominal pain (possible liver or gallbladder problem)
C-chest pain or shortness of breath (possible pulmonaryembolus)
H-headache (possible hypertension, brain attack)
E-ye problems (possible hypertension or vascular accident)
S-severe leg pain (possible thromboembolic process)
OSMOTIC DIURETICS
"GUM"
Glycerol
Urea
Mannitol
OSTEOPOROSIS RISKS
"ACCESS"
Alcohol Use
Corticosteroid Use
Calcium low
Estrogen low
Smoking
Sedentary lifestyle/s
OSTEOPOROSIS RISK FACTORS
“ACCESS” leads to OSTEOPOROSIS
A-alcohol Use
C-corticosteroid Use
C-calcium low
E-estrogen low
S-smoking
S-sedentary lifestyle
PAIN ASSESSMENT
"PQRST”
What Provokes the pain?
What is the Quality of the pain?
Does the pain Radiate?
What is the Severity of the pain?
What is the Timing of the pain?
PAIN MANAGEMENT
Moderate pain (5-6)
Severe pain (7-10) = opioids
PAIN MANAGEMENT
"ABCDE"
Ask about the pain
Believe when clients say they have pain
Choices—let clients know their choices
Deliver what you can, when you said you would
Empower/Enable clients' control over pain
PARKINSON'S DISEASE – CARDINAL SYMPTOMS
"TRAP"
Tremor
Rigidity
Akinesia and bradykinesia
Postural Instability
PARKINSON'S MEDICATIONS
"Ali Loves Boxing Matches"
Amantadine
Levodopa
Bromocriptine
MAO inhibitors
PARKINSON'S MEDICATIONS
"Ali Loves Boxing Matches"
Amantadine
Levodopa
Bromocriptine
MAO inhibitors
PEDIATRIC HYPOXIA SYMPTOMS
"FINES"
Feeding difficulty
Inspiratory Stridor
Nares Flares
Expiratory Grunting
Sternal Retractions
PINPOINT PUPILS
"Drugs, Drops, or Nearly dead"
Drugs: opiates
Drops: meds for glaucoma
Nearly dead: damage in the pons area of the brainstem
PITUITARY HORMONES
"FLAGTOP"
Follicle stimulating hormone
Luteinizing hormone
Adrenocorticotropic hormone
Growth hormone
Thyroid stimulating hormone
Oxytocin
Prolactin
PNEUMONIA – RISK FACTORS
“INSPIRATION”
I - Immunosuppression
N - Neoplasia
S - Secretion retention
P - Pulmonary edema
I - Impaired alveolar macrophages
R - RTI (prior)
A - Antibiotics &cytotoxics
T - Tracheal instrumentation
I - IV drug abuse
O - Other (general debility, immobility)
N - Neurologic impairment of cough reflex, (e.g. NMJ disorders)
PNEUMOTHORAX – S/SX
“P-THORAX”
P - Pleuritic pain
T - Trachea deviation
H - Hyperresonance
O - Onset sudden
R - Reduced breath sounds (&dyspnea)
A - Absent fremitus
X - X-ray shows collapse
POLYCYTHEMIA RUBRA VERA (PRV): COMMON
SYMPTOMS
“PRV”
Plethora/Pruritus
Ringing in ears
Visual blurriness
PORTAL HYPERTENSION FEATURES
"ABCDE"
Ascites
Bleeding (hematemesis, piles)
Caput medusae
Diminished liver
Enlarged spleen
POSTOPERATIVE COMPLICATIONS – ORDER
"4 W's"
Wind (pulmonary)
Wound
Water (urinary tract infection)
Walk (thrombophlebitis)
POST-PARTUM ASSESSMENT
“BUBBLE”
B-breasts
U-uterus
B-bowels
B-bladder
L-lochia
E-episiotomy/laceration/C-section incision
POSTPARTUM ASSESSMENT
"BUBBLE HER"
Breasts
Uterus
Bladder
Bowel function
Lochia
Episiotomy
Homan's sign
Emotional Status
Respiratory System
PREECLAMPSIA(SEVERE) – COMPLICATIONS
"HELLP" syndrome
H- Hemolysis
E- Elevated
L- Liver function tests
L- Low
P- Platelet count
PREGNANCY – INFECTIONS
"TORCH"
Toxoplasmosis
Other (hep B, syphilis, group B beta strep)
Rubella
Cytomegalovirus
Herpes simplex virus
PREGNANCY INDUCED HYPERTENSION
“HELP”
Hemolysis
Elevated liver enzymes
Low
Platelets
PRETERM INFANT – ANTICIPATED PROBLEMS
"TRIES"
Temperature regulation (poor)
Resistance to infections (poor)
Immature liver
Elimination problems (necrotizing enterocolitis [NEC])
Sensory-perceptual functions (retinopathy of prematurity
[ROP])
PROMOTION OF NORMAL ELIMINATION
“POOPER SCOOP”
P-position
O-output
O-offer Fluids
P-privacy
E-exercise
R-report Results
S-size (Amount)
C-consistency
O-occult Blood
O-odor
P-peristalsis
PROSTAGLANDIN USES
"RIPE PI"
Raynaud's
Ischemia of a limb
Pulmonary hypertension
Erectile dysfunction
Peptic ulcers
Induce labor
PROSTATE SYMPTOMS
"no FUN"
Frequency
Urgency
Nocturia
PROSTATE PROBLEMS ARE NO... “FUN”
F- Frequency
U- Urgency
N- Nocturia
PSYCHOTROPIC MEDS – COMMON TCAs
"VENT"
Vivactil
Elavil
Norpramin
Tofranil
PULMONARY EDEMA
“MAD DOG”
M-Morphine
A-Aminophylline
D- Digitalis
D-Diuretics (Lasix)
O- Oxygen
G- Gases (Blood Gases ABG's)
QSEN COMPETENCIES
I
QSEN
PETS
using the I to the N for informatics,
I - informatics
Q - Quality improvements
P - Patient centered care
E - Evidence based practice
T - Teamwork/collaboration
S - Safety
RASHES – DAYS OF APPEARANCE OF
"Very Sick PatientsMust Take Double Exercise"
Varicella(chickenpox)
1st day - Scarlet fever
2nd day - Pox(smallpox)
3rd day - Mumps
4th day - Typhus
5th day - Dengue
6th day - Enteric fever (typhoid)
RASH DAYS AFTER ONSET WHEN IT FIRST APPEARS
"Really Sick Children Must Take No Exercise"
1 Day: Rubella
2 Days: Scarlet fever/ Smallpox
3 Days: Chickenpox
4 Days: Measles (and see the Koplick spots one day prior torash)
5 Days: Typhus & rickettsia (this is variable)
6 Days: Nothing
7 Days: Enteric fever (salmonella)
RENAL FAILURE CAUSES
"AVID GUT"
Acute tubular necrosis
Vascular obstruction
Infection
Diffuse intravascular coagulation
Glomerular disease
Urinary obstruction
Tubulointerstitial nephritis
RESPIRATORY DEPRESSION – INDUCING DRUGS
“STOP” breathing
S - Sedatives and hypnotics
T - Trimethoprim
O - Opiates
P - Polymyxins
RESUSCITATION – BASIC STEPS
“ABCDE”
Airway
Breathing
Circulation
Drugs
Environment
RLQ PAIN DIFFERENTIAL
“APPENDICITIS”
Appendicitis/ Abscess
PID/ Period
Pancreatitis
Ectopic/ Endometriosis
Neoplasia
Diverticulitis
Intussusception
Crohn’s Disease/ Cyst (ovarian)
IBD
Torsion (ovary)
Irritable Bowel Syndrome
Stones
SCARLET FEVER
“6 S’s”
Streptococci causal organism
Sore throat
Swollen tonsils
Strawberry tongue
Sandpaper rash Military
Sudamina vesicles over hands, feet, abdomen
SCHIZOPHRENIA – PRIMARY SYMPTOMS
"4A's"
Affect
Ambivalence
Associative looseness
Autism
SECONDARY HYPERTENSION – CAUSES
“ABCDE”
A:Aldosterone/apnea
B: Bad Kidney/Bruits
C: Catecholamine’s/Cushing’s syndrome
D: Drugs/diet
E: Endocrine
SEVERE PRE-ECLAMPSIA
"HELLP"
Hemolysis
Elevated
Liver function tests
Low
Platelet count
SHOCK – CAUSES
“HAVANA”
Hypovolemia
Adrenal Crisis
Vascular Stasis
Acute Respiratory Obstruction
Neurogenic
Anaphylaxis
SHOCK – SIGNS
“CPR”
Compensatory Stage
Progressive Stage
Refractory Stage
SHOCK
“HYPO-TACHY-TACHY”
Hypotension
Tachypnea
Tachycardia
SHOCK – SIGNS AND SYMPTOMS
“TV SPARC CUBE”
Thirst
Vomiting
Sweating
Pulse weak
Anxious
Respirations shallow/rapid
Cool
Cyanotic
Unconscious
BP low
Eyes blank
SHORTNESS OF BREATH – CAUSES
“AAAA-PPPP”
A - Airway obstruction
A - Angina
A - Anxiety
A - Asthma
P - Pneumonia
P - Pneumothorax
P - Pulmonary Edema
P - Pulmonary Embolus
SICKLE CELL DISEASE
Hydration
Oxygenation
Pain
Infection
Avoid high places
SLE: FACTORS THAT MAKE SLE ACTIVE
“UV PRISM”
UV (sunshine)
Pregnancy
Reduced drug (e.g. steroid)
Infection
Stress
More drugs
SLE SYSTEMIC LUPUS ERYTHEMATOSUS DIAGNOSIS
"MD SOAP 'N HAIR"
Malar rash
Discoid rash
Serositis
Oral ulcer
Arthritis
Photosensitivity
Neurologic abnormality
Hematologic abnormality
ANA (+)
Immunologic abnormality
Renal involvement
SLE SYSTEMIC LUPUS ERYTHEMATOSUS DIAGNOSIS
“DAMP AS RHINO”
Discoid rash
ANA (+)
Malar rash
Photosensitivity
Arthritis
Serositis (pleural, pericardial)
Renal involvement
Hematologic abnormality
Immunologic abnormality
Neurologic abnormality (seizures, psychosis)
Oral / nasal ulcer, Observed
SNS VS PNS
"No sympathy for a Pair of Aces"
Norepinephrine is secreted by the Sympathetic
Acetylcholine is secreted by the Parasympathetic
SPLEEN – CAUSES FOR ENLARGMENT
"3M's"
Myelofibrosis
Malaria
Myelogenous leukemia
SPRAIN – NURSING CARE PLAN
"RICE"
Rest
Ice
Compression
Elevation
SPRAINS AND STRAINS NURSING CARE
"RICE"
Rest
Ice
Compression
Elevation
SSRI – SIDE EFFECTS
“BAD SSRI”
Body weight increase
Anxiety
Dizziness
Serotonin Syndrome
Stimulated CNS
Reproductive/sexual dysfunction
Insomnia
STAPH AUREUS – CAUSES
“SOFT PAINS”
Skin infections
Osteomyelitis
Food Poisoning
Toxic Shock Syndrome
Pneumonia
Acute Endocarditis
Infective arthritis
Necrotizing fasciitis
Sepsis
ST ELEVATION CAUSES IN ECG
"ELEVATION"
Electrolytes
LBBB
Early repolarization
Ventricular hypertrophy
Aneurysm
Treatment (e.g. pericardiocentesis)
Injury (AMI, contusion)
Osborne waves (hypothermia)
Non-occlusive vasospasm
STEROIDS - SIDE EFFECTS
Steroids are an effective treatment for asthma, COPD, Crohn’s, Lupus and more. However, they have the potential for very serious side effects, which are listed below. Steroids can be taken by mouth, via an inhaler, topically, or via injection.
“6 S’s”
Sugar - hyperglycemia
Soggy Bones - causes osteoporosis
Sick - decreased immunity
Sad - depression
Salt - water and salt retention (hypertension)
Sex - decreased libido
STOOL ASSESSMENT
"ACCT"
Amount
Color
Consistency
Timing
SUBARACHNOID HEMORRHAGE (SAH) – CAUSES
“BATS”
Berry aneurysm
Arteriovenous malformation/ Adult polycystic kidney disease
Trauma (e.g. being struck with baseball bat)
Stroke
SYNCOPE CAUSES, BY SYSTEM
"HEAD HEART VESSELS"
-----CNS causes include HEAD:
Hypoxia/ Hypoglycemia
Epilepsy
Anxiety
Dysfunctional brainstem (basivertebral TIA)
-----Cardiac causes are HEART:
Heart attack
Embolism (PE)
Aortic obstruction (IHSS, AS or myxoma)
Rhythm disturbance, ventricular
Tachycardia
-----Vascular causes are VESSELS:
Vasovagal
Ectopic (reminds one of hypovolemia)
Situational
Subclavian steal
ENT (glossopharyngeal neuralgia)
Low systemic vascular resistance (Addison's, diabetic vascular neuropathy)
Sensitive carotid sinus
T AND B CELLS: TYPES
When bacteria enter body, T-cell says to B:
"Help Me Catch Some!"
B-cell replies: "My Pleasure!":
T-cell types:
Helper
Memory
Cytotoxic
Suppressor
B-cell types:
Memory cell
Plasma cell
THERAPEUTIC DOSAGES
"The Magic 2's"
Digitalis (.5-1.5) Toxicity = 2.
Lithium (.6-1.2) Toxicity = 2.
Theophylline (10-20) Toxicity = 20.
Dilantin (10-20) Toxicity = 20.
APAP (1-30) Toxicity = 200.
“THESE DRUGS CAN INTERACT”
“TDCI”
T- Theophylline
D- Dilantin
C- Coumadin
I- ilosone (Erythromycin)
THROMBOTIC THROMBOCYTOPENIC PURPURA: SIGNS
“FAT RN”
Fever
Anemia
Thrombocytopenia
Renal problems
Neurologic dysfunction
THROMOCYTOPENIA: CAUSES
“SHAPIRO”
Splenectomy
Hodgkin's disease
Arteritis
Polycythemia
Infection
Rheumatoid
Occult malignancy
TRACHEOESOPHAGEAL FISTULA ASSESSMENT
"3 C’s"
Coughing
Choking
Cyanosis
TRACTION – NURSING CARE PLAN
"TRACTION"
T-trapeze bar overhead to raise and lower upper body
R-requires free-hanging weights; body alignment
A-analgesic for pain, prn
C-circulation (check color and pulse)
T-temperature (check extremity)
I-infection prevention
O-output (monitor)
N-nutrition (alteration related to immobility)
TRANSIENT INCONTINENCE – COMMON CAUSES
“DIAPPERS”
Delirium
Infection
Atrophic Urethra
Pharmaceuticals
Psychologic
Excess Urine Output
Restricted Mobility
Stool Impaction
TRANSIENT ISCHEMIC ATTACKS ASSESSMENT
"3T’s"
Temporary unilateral visual impairment
Transient paralysis (one-sided)
Tinnitus = vertigo
TRAUMA CARE – COMPLICATIONS
"TRAUMA"
T-thromboembolism; Tissue perfusion, altered
R-respiration, altered
A-anxiety related to pain and prognosis
U-urinary elimination, altered
M-mobility impaired
A-alterations in sensory-perceptual functions and skin integrity (infections)
TRAUMA CLIENT – COMPLICATIONS “TRAUMATIC”
T-tissuePerfusion Problems
R-respiratory Problems
A-anxiety
U-unstable Clotting Factors
M-malnutrition
A-altered Body Image
T-thromboembolism
I-infection
C-coping Problems
TRAUMA SURGERY (“AMPLE”) AFTER INITIAL ASSESSMENT
A-allergies
M-medications
P-past Medical History
L-last Meal
E-events Surrounding Injury
TRAUMA SURGERY PREP
"AMPLE"
Allergies
Medications
Past Medical History
Last Meal
Events Surrounding Injury
TYPES OF INCONTINENCE
"OUTS"
Overflow incontinence (loss when specific volume
reached)
Urge incontinence (loss from contraction that follows strong, sudden need)
Total incontinence (complete loss of urinary control, as from a nonfunctioning urethral sphincter muscle)
Stress incontinence (loss of small amounts of urine when abdominal pressure increases through cough, sneeze, or laugh)
URINARY INCONTINENCE CAUSES
"DRIP"
Delirium
Restricted mobility, retention
Infection, inflammation, impaction
Polyuria, pharmaceuticals
VENTRICULAR FIBRILLATION – TREATMENT
"Shock, Shock, Shock, Everybody Shock, Little Shock, Big Shock, Momma Shock, Poppa Shock"
Shock= Defibrillate
Everybody= Epinephrine
Little= Lidocaine
Big= Bretylium
Momma= MgSO4
Poppa= Procainamide
VFIB/VTACH – DRUGS USED ACCORDING TO ACLS
"Every Little Boy Must Pray"
Epinephrine
Lidocaine
Bretylium
Magsulfate
Procainamide
VIRCHOW'S TRIAD: FACTORS LEADING TO VENOUSTHROMBOSIS
"VIR-chow"
Vascular trauma
Increased coagulability
Reduced blood flow (stasis)
WARFARIN INTERACTIONS
"ACADEMIC QACS"
Amiodarone
Cimetidine
Aspirin
Dapsone
Erythromycin
Metronidazole
Indomethacin
Clofibrate
Quinidine
Azapropazone
Ciprofloxacin
Statins
WBC RELATIVE CONCENTRATIONS
"Never Let Monkeys Eat Bananas" (From greatest to least)
Neutrophils 56%
Lymphocytes 34%
Monocytes 4%
Eosinophils 2.7%
Basophils 0.3%
• Alternatively: "Nine Little Monkeys Eating Bananas"
• Alternatively: "No-one Likes My Educational Background"
• Alternatively: "Never Let Mamma Eat Beans"
• Can remember that Eosinophils is 3%, by the mirror image of E is 3.
WBC – In order of decreasing numbers
“Never Let Monkeys Eat Bananas”
Neutrophils
Lymphocytes
Monocytes
Eosinophils
Basophils
[Show More]