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NURS 3320 Patho 1 Final Exam Blueprint| Comprehensive for Grade A+

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Mobility • Know and understand the structural changes in scoliosis and the common complications observed in this population. ▪ A lateral deviation of the spinal column commonly in the coronal or frontal plane that may ...

Mobility • Know and understand the structural changes in scoliosis and the common complications observed in this population. ▪ A lateral deviation of the spinal column commonly in the coronal or frontal plane that may or may not include rotation or deformity of the vertebrae. ▪ A three-dimensional deformity of the spine, often affects the entire skeletal torso. ▪ It can be classified with regard to age of onset and etiology. Generally girls are affected. ▪ The majority of scoliotic deformities are idiopathic. Others can be congenital, associated with neuromuscular disorders (cerebral palsy or muscular dystrophy), a result of intraspinal deformities, or caused by uneven lower limb length. ▪ Usually is first noticed because of the deformity it causes ▪ A high shoulder, prominent hip, or projecting scapula may be noticed by a parent or in a school-based screening program. ▪ Difficulty in hemming or fitting a dress may call attention to the deformity. ▪ Idiopathic scoliosis usually is a painless process, although pain may be present in severe cases usually in the lumbar region. The pain may be caused by pressure on the ribs or on the crest of the ilium. ▪ There may be shortness of breath as a result of diminished chest expansion and gastrointestinal disturbances from crowding of the abdominal organs. ▪ Adults with less severe deformity may experience mild backache. If scoliosis is left untreated, the curve may progress to an extent that compromises cardiopulmonary function and creates a risk for neurologic complications. • Understand the structural changes inside the joint with osteoarthritis o Progressive disruption of the smooth surface of the articular cartilage with development of surface cracks that deepen to involve the subchondral bone, followed by complete erosin of the articular cartilage with exposure of ivory-like polished subchondral bone, dislodgment of fragments of free-floating osteocartilaginous bodies, development of bone cysts, and formation of abnormal bony spurs at the joint margins

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